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The mTORC1 pathway stimulates glutamine metabolism through the c-Myc transcription factor, which controls the expression of genes required for glutaminolysis, such as glutaminase 2 and other enzymes in c-Myc-stimulated T-cells (72)

In diabetic mice, xCT inhibited the release of the TGF- family member GDF15 from DCs, resulting in slow wound healing

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