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Furthermore, a separate investigation into mitophagy in RA has demonstrated that MST1 suppresses the SIRT3/mTOR signaling, leading to attenuated mitophagy and enhanced mitochondrial fission, thereby reducing inflammation, proliferation, and invasiveness of RA-FLSs (61)

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Increasing evidence indicates that the transition to CKD is accompanied by persistent decreases in fatty acid -oxidation with renal tubular cell atrophy but continued high expression of glycolytic enzymes [52]