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BBB impairment, including reduced expression or weakening of tight junctions, is responsible for the disruption of BBB integrity in PD, leading to increased vascular permeability and the accumulation of LBs, proteins and other intermediates, such as oligomeric -syn in neuronal cells, the basal ganglia and activated astrocytes from the SNpc [172, 173]

El Achi, H., Dupont, E., Paul, S

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The study highlights that oxidative stress is not merely a byproduct of these illnesses, but a core, shared mechanistic driver of the pathology