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Proinflammatory mediators in the bronchoalveolar lavage fluids from oxidant-expose lungs (CEES caused a 20-fold, fourfold, and 1.6-fold increase in CINC-1, IL-1, and TNF-, respectively) were reduced to baseline levels [99]

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As a key mediator of innate immunity, the NLRP3 inflammasome contributes to immune regulation

It is necessary to understand the role of catalase in disease-specific contexts, paving the way for precision antioxidant therapy that could significantly impact the treatment of diseases associated with oxidative stress

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