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Nukui et al., 2006

Any of these structures are prone to injury due to the frequency that the hips are used

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Aside from these examples, little is known about the biophysical impact of many other sitespecific posttranslational modifications on apoSOD1 stability, despite numerous solventaccessible sites for glutathionylation, acetylation, glycation, phosphorylation, succinylation and ubiquitylation (Figure 9).[ 62 , 72 , 86 , 120b , 124 ] Advances in this area will require greater refinement of emerging methodologies designed to target certain modifications to specific protein residues in vitro and in vivo, [132] including selective chemical modification of proteins, protein semisynthesis, native chemical ligation, and the genetic noncanonical amino acid incorporation approach

Results Design of an endogenous protein domain-based haemostatic protein During haemostasis platelets facilitate fibrin gel formation at injury sites by recognising exposed collagen, recruiting fibrinogen and initiating thrombin-mediated fibrin polymerisation (Fig

Journal of Infectious Diseases, 156(2), 206210