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doi:10.1002/ibd.21588 Hrubisko, Martin et al

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A truncated 67-aa IGF-1 analog lacking the N-terminal Gly-Pro-Glu tripeptide, with higher intrinsic IGF-1R affinity and moderately reduced IGFBP binding

Silibinin decreases prostate-specific antigen with cell growth inhibition via G1 arrest, leading to differentiation of prostate carcinoma cells: implications for prostate cancer intervention