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MOTS-c is not simply released from the mitochondria and degraded locally

Continued interdisciplinary collaboration is essential to bridge the gap between exciting benchtop discoveries and tangible bedside applications, ultimately fulfilling the promise of personalized nanomedicine for respiratory patients

PRDX6 selectively binds oxidized phospholipids, becomes membrane-associated, and executes coordinated membrane repair through reductive detoxification, iPLA 2 -mediated deacylation, and LPCAT-dependent reacylation with non-oxidized fatty acids

Perni M, Mannini B

This process highlights the dual role of ROS in tumors 76 : while they may promote tumor evolution under specific conditions (e.g., inducing DNA damage, facilitating angiogenesis, and activating immunosuppressive pathways), they may also exert anti-tumor effects by triggering cell cycle arrest

Occurs due to genetic or epigenetic factors, or in response to toxins, inflammation, and oxidative stress