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One of the most prominent metabolic features in prostate cancer is the activation of de novo fatty acid synthesis [2,3,4], in which acetyl-CoA carboxylase alpha (ACACA) serves as a key rate-limiting enzyme [5, 6]

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This increased anticoagulant effect from warfarin may be due to a combination of pharmacokinetic and pharmacodynamic factors

doi: 10.1002/hep.23696 6 FontanaRJHayashiPHGuJReddyKRBarnhartHWatkinsPBet al

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In some embodiments, the method further comprises coupling a peptide epitope to the peptide of the peptide conjugate