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In summary, GSH enables antibiotic inactivation by serving as a substrate for detoxifying enzymes, such as FosA, and by modulating the expression or stability of antibiotic-degrading enzymes, including the downregulation of NDM-1

ALA also shows improving acetylcholine levels in synapses

van Eijk LE, Offringa AK, Bernal ME, Bourgonje AR, van Goor H, Hillebrands JL

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2021 Jan 1;116(1):15261

4 Anti-rheumatic drugs and ferroptosis Using ferroptosis inducers to inhibit tumor progression is currently a popular direction in the field of tumor therapy