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Cancer Cell 2004

Whether the mitochondrial dysfunction in RA can be associated with ferroptosis and what kind of ferroptotic cells should be linked to the mitochondrial dysfunction in RA still need further research and exploration

In contrast, the negative effects of deficiency on the mitochondrial function, cholesterol metabolism, synthesis of steroid hormones (sex hormones) and formation of carcinogenic nitrosamines are relatively well comprehended

Such damage can further lead to mitochondrial dysfunction, triggering apoptosis or necroptosis, which represents a key pathological process in oxidative stress [61]

Ramadan et al., 2022

Molecular chaperone peptides B-crystallin (HSPB5) and its peptide derivatives, studied to protect dermal proteins against aggregation and oxidative damage in photoageing