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Description

Vandrey, R., Raber, J

We have generated the McGill-R-Thy1-APP transgenic (Tg) rat model exhibiting an AD-like amyloid pathology with a prolonged pre-plaque stage, which allows for studying the effects of gradual iA accumulation well before extracellular plaque formation 12,14,36

Starch grains are positioned close to the border of the pyrenoid matrix (Krienitz et al

In this context, ROS-triggered TP53 activation heightens mitochondrial membrane permeability, leading to the release of pro-apoptotic factors from mitochondria

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