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Description

Immune Dysfunction and Infections in Patients With Cirrhosis

Proteins were transferred to PVDF membranes using transfer buffer, and immunoblotted for parkin, DJ-1, MnSOD, Aco2, mitochondrial creatine kinase (mtCK), VDAC, TOM20, nitrotyrosine, glutathione reductase, and glyoxalase-1

doi: 10.1038/srep04089

However, if the right ventricular compensation threshold is surpassed, it can result in right ventricular hypertrophy, chamber dilation, fat deposition, fibrosis [3], and ultimately lead to death from right heart failure