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These include anemia, neurological issues, and a higher risk of heart disease

Available evidence suggests that the occurrence of HACI involves multiple interacting pathological processes, in which aggravated oxidative stress and neuroinflammation have been repeatedly reported and are considered to be one of the key mechanisms involved (Shushanyan et al., 2024

However, roughly 25% of people who survive a ruptured brain aneurysm have cognitive deficits or discrete neurologic deficits like difficulty speaking, weakness, and other symptoms stemming from permanent brain damage