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Alloying represents a powerful approach to retune the electron distribution and geometry of nanozymes, thereby achieving excellent catalytic performance

Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

Levels of 0.40% or higher are potentially fatal, with risks including coma and death due to respiratory arrest

Intended for research use only in qualified laboratory settings

Think of it as a trade-off between known side-effect profiles and unknown long-term effects