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FOXO4-DRI has been suggested to prevent normal FOXO4 binding to p53, eliminate senescent cells, enhance organ function, and reduce cell death progression

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Upon binding of the ligand, PPARs heterodimerize with retinoid X receptors and bind to cis-acting DNA elements (PPREs) to increase gene transcription

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Taken together, these findings underscore the critical role of SIRT1 in modulating inflammation-driven ferroptosis through NF-B, NLRP3 and AMPK signaling