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Goenjian AK, Yehuda R, Pynoos RS, Steinberg AM, Tashjian M, Yang RK et al (1996)

Recent findings demonstrating the key role of fatty acid oxidation for renal cell proliferation and compensatory hypertrophy may help to understand the complex mechanisms involved in this process and whether abnormal lipid metabolism in the injured kidney may contribute to IGF-1 resistance

The findings are summarized in Table 2

The pathway may still be active, but may nonetheless not be necessary for the endpoint being investigated

Symptoms during the first 24 hours are not diagnostic or specific and include nausea, vomiting, diaphoresis, anorexia, and lethargy, the severity of which will be in direct correlation to the size of the dose ingested

[DOI] [PMC free article] [PubMed] [Google Scholar] 10.Kleinstern G, Larson DR, Allmer C, Norman AD, Muntifering G, Sinnwell J, et al