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Thus, functional deletion of GluN2A renders PV interneurons susceptible to an oxidative insult during their critical period of maturation leading to long-lasting PV neuron/PNN anomalies and reduced high-frequency neuronal synchrony that are prevented by NAC [130]

Dalziel, K

L-carnitine and D-carnitine, but it is known that only the L-isomer of carnitine is biologically active

In a further study of the mechanism of ferroptosis on inflammasomes, after applying Fer-1 and erastin, the expression of NLRP3, NLRP1, IL-1, and caspase-1 increased, indicating a mutually restrictive relationship between ferroptosis and the NLRP1 inflammasome (182)

This, in turn, resulted in abnormal glycolysis and increased intracellular glucose levels (up to 75%) (Fig 4F), which also confirmed the results of the omics data analysis