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Care 11 , R114 (2007)

Inflammation Research , 57(5), 205-210

The molecular mechanisms proposed to explain how ROS could specifically mediate brain damage are the following: (1) The lower levels of antioxidants can promote increased activity of lipoxygenase in CNS stimulating leukotriene production, thereby increasing the immunoinflammatory processes in the cerebral cortex

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[] Glutamate concentrations in the left ventrolateral PFC changed from the starting point and MR spectroscopy showed a strong correlation between the decrease of complaints and this change