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The extract also showed inhibition on the release of inflammatory enzymes (COX-2, iNOS, and MMP-9) by inhibiting LPS-induced activation of JAK/STAT and TLR4 pathways, particularly JNK, p38, IB, and STAT phosphorylation

Furthermore, IGF1R silencing mimicked the tumor-suppressive effects of miR-532 in CRC cells, further confirming its direct interaction with IGF1R, and miR-532 overexpression inhibited PI3K/AKT pathway activation both in vitro and in vivo (92)

Lewis, K

The Interplay Between Stress, Inflammation, and Emotional Attention: Relevance for Depression

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