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In vitro and in MIA induced murine OA, the system promoted macrophage polarization from M1 to M2, reduced proinflammatory cytokines, shifted the joint milieu toward an anti-inflammatory state, preserved chondrocyte metabolic homeostasis, and supported BMSC chondrogenic differentiation (Fu et al., 2025) (Figure 2D)

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Supplementary Table 2 (download XLSX T cell surfaceomes labeled by surface-targeted LaccID

163 In addition, Tfh cell production and function are dependent on Notch signaling, Notch receptors 1 and 2 are required for Tfh cell production, and Notch signaling enhances the production of type 2 cytokines in Tfh cells