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Ebselen stabilizes mutant SOD1 in living cells Several ALS-linked SOD1 mutants fail to bind zinc in the human cytosol, and accumulate as unstructured species that may act as precursors in the pathogenic aggregation of SOD1 8,9

Collectively, these findings demonstrate forskolins capacity to address multiple pathological features of AD, including amyloid plaque accumulation, tau hyperphosphorylation, oxidative stress, neuroinflammation, and synaptic dysfunction

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There is growing evidence that various neurodegenerative diseases, including Parkinsons disease, stroke, Huntingtons disease, and traumatic brain injury, are associated with ferroptosis (98, 99)