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The final pathological manifestation of CKD is renal fibrosis, characterized by glomerulosclerosis, renal tubular atrophy, and interstitial fibrosis [223, 224]

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These approaches include combinatory administration of antibiotics, strengthening colonisation resistance of the gut microbiota to reduce VRE proliferation through commensals or probiotic bacteria, or switching to non-antibiotic bacterial killers, such as bacteriophages or bacteriocins

It also works by suppressing noradrenaline which results in dilation of the cavernosal arteries resulting in engorgement of the pens which leads to an erection