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This indicates that epigenetic changes in mtDNA, especially in the MT-RNR1 region, mediate the relationship between PM2.5 exposure and reduced mtDNA content, suggesting mechanisms of mitophagy and mitochondrial dysfunction in the early life environment

Ultrathin sections were cut at thicknesses of 6070 nm with an ultramicrotome (Leica, Germany) and stained with uranyl acetate and lead citrate

Treatment with either the EAAT1 or EAAT2 antisense oligonucleotides caused a progressive motor impairment whereas the EAAT3 antisense oligonucleotide mainly produced epilepsy