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Genetic variation in Mon1a affects protein trafficking and modifies macrophage iron loading in mice

This is mechanistically different from injecting recombinant GH itself: a GHRH agonist raises GH indirectly, through the pituitary, so the output remains at least partly subject to the axiss normal regulation including negative feedback from IGF-1 and somatostatin

Since TC and TG are independent risk factors of in DN (Jansson Sigfrids et al., 2021

Indeed, our findings could suggest that AcM may have led to the selection of a functional genetic potential less prone to PGN synthesis, possibly due to the selection of species with less active PGN turnover

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