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However, antioxidant treatment (histamine dihydrochloride and diphenylene iodonium chloride) partially restored the ADCC effect of NK cells (235)

Understanding the genetic, ecological, and physiological factors underlying this refractoriness is crucial for designing targeted interventions

Since we observed an activation of NRF2 upon GSH depletion, we expected NRF2 to be essential for preventing liver damage

Further oxidation of Hz and AcHz by CYP450 enzymes, particularly CYP2E1, generates reactive intermediates that form covalent adducts with endogenous macromolecules, a process implicated in INH-induced hepatotoxicity (Delaney and Timbrell, 1995

[DOI] [PubMed] [Google Scholar] 21.Gelosa P., Colazzo F., Tremoli E., Sironi L., Castiglioni L

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