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Our observations of a concentration-dependent inhibition of NFT-dependent glutathione synthesis stimulation (Fig

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BioMed Pharmacother (2019) 118:109249

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Furthermore, we now have evidence that mutations in TP53 and EGFR attenuate ROS accumulation in cancer cells by sustaining an oncogenic oxidant intracellular environment through an integrated regulation of redox-related enzymes and signaling pathways (e.g