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Puf-A plays an important role in a mouse model of mutant Kras/p53 loss driven lung ADC To confirm the in vivo interactions between Puf-A and p53, we employed a previously established lung cancer model using CCSP-rtTA/TetO-Cre/LSL-Kras G12D /p53 +/+ mice [27], which developed lung adenoma, followed by ADC at 6 and 12 weeks, respectively, after Kras G12D induction (Fig

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