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Senescent cells, despite their pro-inflammatory SASP, also suppress tumor progression in some contexts through 'senescence surveillance'nearby immune cells recognize and destroy newly senescent cells, including pre-malignant cells undergoing oncogene-induced senescence

This characteristic makes it potentially safer for long-term applications, as elevated IGF-1 levels associated with traditional GH therapy raise concerns about increased cancer risk and metabolic complications

At Rivas Medical, for example, when we first started using semaglutide we found patients reported more side effects when they jumped from 0.5mg to 1.0mg per week

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What exists is a real, documented biological mechanism that offers a plausible explanation, and a much larger body of anecdotal reporting built on top of that mechanism

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