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NIH/PMC research on GHK as a cellular pathway modulator describes how GHK-Cu induces systemic wound healing in rats, mice and pigs when injected at one site, improving healing at distant body areas, increasing collagen production, angiogenesis and wound closure parameters across both wound chambers and full thickness wounds

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Reactions don't improve with continued use

G protein-coupled inwardly rectifying K+ channels (GIRKs) mediate postsynaptic but not presynaptic transmitter actions in hippocampal neurons

CS exposure significantly extended Lm when compared to control group

Some clinics use an intranasal formulation