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Similar content being viewed by others Introduction Glutathione (GSH) deficiency is one of the earliest biochemical perturbations in Parkinsons disease (PD), 1,2 leading to the hypothesis that GSH supplementation may have therapeutic value in alleviating PD symptoms or modifying progression
Long term GSH supplementation led to notable changes in the diabetic gut microbiome
Nevertheless, current GSH-activated prodrugs frequently face challenges in achieving selective activation within heterogeneous tumor microenvironments
Direct stimulation of K(ATP) channels by exogenous and endogenous hydrogen sulfide in vascular smooth muscle cells
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