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Description

Rather than acting as a classic receptor agonist with a single defined binding site, BPC-157 appears to influence multiple regulatory nodes involved in endothelial signaling and cellular stress response pathways

Continuous oxidation and reduction of these SS is possible due to fast and reversible electron transfer between the active site cysteines of thiol-redox enzymes and the cysteines in the target protein [130]

The FDA explicitly advises against it

Supportingly, a reduction of reserpine-hypothermia (NaBPC157 pre-treatment) and reversal of further prominent temperature fall (NaBPC157 post-treatment) have been consistently observed