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Telomerase activity, mediated by TERT expression, declines in an age-dependent manner, and the ratio of telomere length to telomerase activity provides a molecular measure of cellular aging that complements traditional ovarian reserve markers [192, 216]

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the method of the invention comprises administering to a subject a composition comprising an EphA2 or EphA4 targeting moiety attached to a therapeutic or prophylactic agent against the hyperproliferative cell disease

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Additionally, functionality was restored to the quadriceps muscle tissue

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It would appear that mitochondrial dysfunction and reactive oxygen species development play an important role in the progression from simple steatosis to steatohepatitis in ALD (134, 135)

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Studies on wound healing models reveal rapid gene expression changes and angiogenic activity in laboratory settings[1][5]

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