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Under normal conditions, microglia maintain neuronal function through synaptic pruning, but in early AD, hyperactive neurons trigger aberrant microglial phagocytosis, causing synaptic loss and disrupted neural signaling (Zhong L

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Only the NCM derived from the N-terminus TNC (1700aa) expressing cells showed significantly rescued pro-MuSC-proliferation capacity, while NCM derived from the other truncated TNC-expressing cells failed to promote MuSC proliferation (Supplementary information, Fig