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The primary injury results in localized brain damage, while the secondary damage initiates inflammatory responses, followed by the disruption of the blood-brain barrier, infiltration of peripheral blood cells, brain edema, and the release of various immune mediators, including chemotactic factors and interleukins

Mares A, Miah AH, Smith IED, Rackham M, Thawani AR, Cryan J, et al

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Our findings show that the depletion of GSH by TNBS is a necessary but insufficient event by itself to cause injury