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Zhao et al (184) found that AF can markedly inhibit -SMA overexpression and FN deposition in the livers of rats treated with BDL and HSC activation and collagen I and III synthesis are also markedly inhibited by coadministration

Notably, bell-shaped dose-dependent profiles were again observed in these experiments, consistent with the aforementioned doseresponse studies

Abstract Liver diseases, including hepatocellular carcinoma (HCC), non-alcoholic fatty liver disease (NAFLD), and alcoholic liver disease (ALD), impose a significant global health burden, with over 2 million deaths annually and substantial economic losses

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