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Generation of superoxide anion by the NADH dehydrogenase of bovine heart mitochondria

Electrophysiological and phenotypic profiles of taxane-induced neuropathy

On the other hand, oxidative stress leads to an increase in the release of cytochrome C from the mitochondria which activates the apoptotic mechanism in the cells by activating caspases (caspase-3/7, 8 and 9) and pushing the cells to the programmed cell death

By facilitating the entry of fatty acids into the mitochondria, L-carnitine ensures that these molecules are efficiently utilized for energy production, particularly in skeletal muscle and the heart

The total phenolic content was expressed as milligrams of gallic acid equivalents (mg GAE/kg dry matter), calculated based on a standard curve generated from gallic acid solutions of known concentrations [42]

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