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We previously found that cancer cells in a high mesenchymal therapy-resistant cell state are dependent on the lipid hydroperoxidase GPX4 for survival 6

Each method occupies a specific niche in the preclinical research workflow

To further validate the functional importance of the secreted form of OLFM4, we reconstituted OLFM4-knockdown HGC-27 cells with wildtype or DelSP mutant OLFM4 to either restore both intracellular or secreted function of OLFM4 or only restore its intracellular function

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Researchers have found that DSIP can actually reduce stress-induced changes in animal models

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