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*Correspondence: Huan Chen, [email protected]

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Many of these trials recruited terminal or end-stage patients, for whom any sort of treatment is unlikely to have an effect, and they often recruited cohorts with mixed cancers, which may respond differentially to IVC depending on the underlying mechanisms involved

doi: 10.1038/s12276-022-00736-w 89 YamazakiTHannaniDPoirier-ColameVLadoireSLocherCSistiguAet al

Maps sharpened less aggressively ( B -factor of 15.0 2 ) confirmed unambiguously that this feature was due to the presence of CP17 bound at the interprotomer pocket of the wt PV2 VLP (Fig

How does hepatic lipid accumulation lead to lipotoxicity in non-alcoholic fatty liver disease