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GPX4, a selenocysteine-containing enzyme, utilizes GSH to reduce toxic phospholipid hydroperoxides (PL-OOHs) to non-toxic phospholipid alcohols (PL-OHs), thereby disrupting lipid peroxidation chain reactions and preventing oxidative cell injury (Pontel et al., 2022)

Thus, glutamatergic dysfunction in schizophrenia is certainly associated to metabolic impairments

(2021) 131:e141529

As an alternative to the H3K4-methylation-dependent mechanism that mediates up-regulation of p65/RelA expression, Li et al

doi: 10.1016/j.neulet.2011.08.047

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