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Mitochondrial protection is exerted by GSH versus radicals and oxidant species by the contribution of a group of nutrients that can directly or indirectly protect mitochondria from oxidative damage and improve mitochondrial function [17]

However, when SELENOP was directly targeted, we found that children with all stages of the disease did show a statistically significant reduction in signal as compared to their unaffected sibling

In the zebrafish liver cancer model, YAP reprograms glutamine metabolism by enhancing de novo nucleotide biosynthesis and regulates GS expression to induce cell proliferation 9

Bohle B, Radakovics A, Jahn-Schmid B, Hoffmann-Sommergruber K, Fischer GF, Ebner C

[10] According to a systematic review, we can see that the variations in G6PD activity are significantly different among studies due to different ethnic groups and populations

Payne DJ, Gwynn MN, Holmes DJ, Pompliano DL