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Pretreatment of HaCaT cells with an IP3R inhibitor indicates that GSK3 may regulate the activation of IP3R, and thereby modulate the release of Ca 2+ from the endoplasmic reticulum into the cytoplasm, thus indirectly regulating mitochondrial damage, and apoptosis

[224] and others [225] have reported that providing leucine after completion of resistance exercise can further potentiate increases in mTOR signalling and protein translation

(en) Exner R, Wessner B, Manhart N, et al

The efficacy comparison of classic ketogenic diet and modified Atkins diet in children with refractory epilepsy: a clinical trial

J.TangH.SunP.et al (2013a)

however, in vivo, one could imagine that repeated 5FU exposures would drive selection of pre-existing niches of tumours cells that had higher mitochondrial functionality