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Acknowledgements We would like to acknowledge support from Fondos Federales (Grant number HIM 2019/029 SSA 1575)

Discov Med 16:5363 Franklin RJM (2002) Why does remyelination fail in multiple sclerosis

Bioeng Biotechnol

Our findings reveal an unexpected role for GPX2 in modulating pancreatic and hepatic cell fate decisions and provide insights into the intricate interplay between oxidative stress and cell fate determination during hPSC differentiation into endoderm derivatives, with emphasis on pancreatic endocrine cell development

26 H 2 O 2 is not a very oxidizing molecule but can yield the highly reactive hydroxyl radical (HO ) by reduction by metals (Fenton reaction) (eq 3) (Figure 1)

Knockout of prolyl hydroxylase domain-containing protein 2 (PHD2) stabilized HIF-1 protein and activated HIF-1 signaling, which transcriptionally upregulated glutaminase 1 ( GLS1 ) expression leading to a decrease in ROS production and increases in cellular GSH levels, GSH/GSSG, and cell survival in murine periosteal progenitor cells, suggesting that HIF-1 signaling promotes glutaminolysis to provide glutamate for GSH synthesis (96)