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doi:10.1097/AJP.0b013e318255655d

Studies focus on how Cagrilintide modulates alpha cell activity during postprandial states to reduce inappropriate glucagon secretion, helping to maintain homeostatic hepatic glucose output under experimental conditions

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however, the negative charge at the C terminus and its long range non-covalent interaction with the N terminal domain protects the protein from losing its native state and committing to fibrillation