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To view a copy of this licence, visit About this article Cite this article Baquero-Prez, B., Yonchev, I.D., Delgado-Tejedor, A

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shows that 7nAChRs knockout mice that do not respond to cholinergic agonists, which in turn enhance the production of several cytokines and inflammatory markers, including tumor necrosis factor-alpha (TNF-) and interleukin-1 (IL-1) [65], this results from suppression of signal transduction cascade including PIK3/Akt and Nrf2 and upregulation of NF-B that represent a transcriptional factor capable of coordinating the inflammatory response, regulating the inflammatory gene expression [35, 66]

(7) L Pickart, J Michelle Vasquez-Soltero, and A Margolina (2012) The Human Tripeptide GHK-Cu in Prevention of Oxidative Stress and Degenerative Conditions of Aging: Implications for Cognitive Health Oxidative Medicine and Cellular Longevity, 2012 (Issue 1), Page 324832

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