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Circadian control of the NAD + salvage pathway by CLOCK-SIRT1

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These results indicate that the adipose tissue is one of the main sources of secreted extracellular NAMPT (eNAMPT)

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DNA damage-induced PARP1 activation confers cardiomyocyte dysfunction through NAD(+) depletion in experimental atrial fibrillation

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PMID 28756557

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