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Although moderate levels of ROS act as signaling molecules in cellular processes such as differentiation, proliferation, and adaptive responses [153], excessive ROS accumulation induces lipid peroxidation, protein modification, and mitochondrial DNA damage, ultimately leading to cellular senescence, apoptosis, or dysfunction [154]

J Toxicol Pathol 11, 3340

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It is worth noting that ferroptosis inhibitors have been shown to exacerbate the progression of GC, further underscoring the significance of targeting ferroptosis in GC treatment

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Free Radic Biol Med 62, 111120 (2013)