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Additionally, ROS can directly oxidize and modify key autophagy modulators, such as ATG4, facilitating the formation of autophagosomes [165]

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Bradley JD, Hu C, Komaki RR, Masters GA, Blumenschein GR, Schild SE

doi: 10.1111/j.1471-0528.1964.tb04317.x 62

These observations were also made with mechanistically different secondary stressors such as the mitochondrial complex I inhibitor, rotenone, or proteasomal inhibitors such as MG-132 or bortezomib (Suppl

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