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doi: 10.1093/hr/uhae043 10 ChandraS

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10.1155/2020/3635787 160 QiangW.ShenT.NomanM.GuoJ.JinZ.LinD.et al (2021)

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RNA-Seq was performed in activated T cells, and GSEA plot showed the enriched pathway of oxidative phosphorylation in the combination of both compared with single treatment alone ( A )

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ALA effectively stabilizes hepatic redox-active iron (Fe 2+ ) expression and depresses the formation of ferroptosis after CSD Considering that overload of the redox-active iron (Fe 2+ ) plays a harmful role in hepatic metabolic function due to its involvement in catalyzing the Fenton reaction, generating ROS, enhancing oxidative stress, exacerbating inflammatory signaling, and leading to the development of ferroptosis, the present study further examined the hepatic Fe 2+ expression, together with the ferroptotic related proteins by the use of TOF-SIMS analysis and immunohistochemistry, respectively

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Zhang, Dual Function of Magnesium in Bone Biomineralization

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