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More recent studies have focused on specific cellular actors, showing that metabolites from different bacterial species can regulate enterocyte lipid metabolism [62]

Research has demonstrated that vitamin B5 facilitates mitochondrial respiration and metabolic maturation, and exerts a critical regulatory effect on intracellular energy homeostasis (55)

Interestingly, this regulation was not observed in hepatocellular carcinoma HepG2 cells, suggesting cell-type-specific mechanisms and potential vulnerability of cancer cells to sterol metabolite modulation [17]

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