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POS has been proposed as a probable mechanism contributing to systemic OxS, T2D progression, and vascular damage (see Figure 3) [91,92]

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Kamkaew N, Paracha TU, Ingkaninan K, Waranuch N, Chootip K

Lipid peroxidation seems to be promoted by cell cycle arrest, as it was not observed in palbociclib resistant cell lines with an RB1 loss or CCNE1 amplification